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Food poisoning

Xem 1-20 trên 24 kết quả Food poisoning
  • The result pointed out that only 5 g of soft tissue from N. glans (equivalent to 2–3 specimens) containing maximum toxicity detected in the present study may cause human death if consumed. Fhe first time, this study identified TTXs in the gastropods as a causative toxin in the poisoning in Vietnam. Moreover, 65.5% of studied specimens with high toxicity higher than 100 MU/g, including 16.1%, showing extremely high toxicity (> 1,000 MU/g). The results suggested that this gastropod is quite dangerous for human consumption and should be alerted to public awareness.

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  • For this study, disease infected leaf samples were collected from pulse pathology block situated at Normon E. Borlaug Crop Research Centre, Pantnagar based on cultural, morphological and disease potential studies and then evaluated the pathogen against five botanicals viz. Calotropis, Ocimum, Aloevera, Neem and Bael at 5, 15, 25 and 35 per cent concentration through poison food technique and two bioagents viz.

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  • In the present investigation 30 Plant leaf extract were evaluated in in vitro condition against P. palmarum adopting poisoned food technique. The per cent inhibition of pathogen was 100 per cent by Dhatura, Anjwain and Tobacco at 10 per cent concentration followed by the turmeric (95.84), safed musli (90.62), garlic (87.5), hathjodh (86.5), kalmegh (86.46), jetropha (83.34), neem bark (79.18), satavar (78.12), lemongrass (69.81), laung (67.71), ashoka (65.62), aadusa (63.68), karanj bark (63.06), bhringraj (62.5), karanj (62.5), dalchini (61.15), beshram (60.43), brijdanti (57.

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  • Foot rot of tomato is an important disease which causes severe loss in tomato production. The present study was carried out to evaluate five bioagents and six fungicides against Sclerotium rolfsii. Among the five antagonists tested the maximum inhibition was observed in Trichoderma virens (67%) and contrarily, no inhibition was observed in Bacillus subtilis. Six fungicides viz., carbendazim, captan, tebuconozole, copper oxychloride, hexaconozole and Tebuconazole +Trifloxytrobin at different concentrations (50, 100, 150, 200, 250 and 500 ppm), were tested.

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  • Curriculum "English for Nutrition and Food Siences: Part 2" continue to introduce to you the information, knowledge:  Unit 6 - Human Diet, Unit 7 - Food Poisoning, Unit 8 - Organic Food , Unit 9 - Genetically Modified Food , Unit 10 - Food Industry. Invite you to refer to capture the details of the curriculum.

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  • Ngộ độc thực phẩm (Food poisoning) là các biểu hiện bệnh lý xuất hiện sau khi ăn uống những loại thực phẩm nhiễm khuẩn, nhiễm độc hoặc có chứa chất gây ngộ độc. Bài viết này sẽ cung cấp cho bạn đọc những kiến thức cơ bản về định nghĩa, phân loại ngộ độc thực phẩm, nguyên nhân chủ quan và nguyên nhân từ các tác nhân gây ngộ độc, quá trình và các triệu chứng của ngộ độc thực phẩm, phương pháp phòng và phương pháp điều trị dành cho người bị ngộ độc.

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  • Vibrio mimicusis a causative agent of human gastroenteritis and food poisoning, and this species produces an enterotoxic hemolysin (V. mimi-cus hemolysin) as a virulence determinant. Vibrio mimicushemolysin is secreted as an 80 kDa precursor, which is later converted to the 66 kDa mature toxin through removal of an N-terminal propeptide via cleavage of the Arg151–Ser152 bond.

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  • Water used in slaughtering operations are considered one source of infected carcass during slaughter, particularly in the private slaughterhouses. The purpose of this study was to identify some species of microorganisms causing food poisoning from the water samples used in the slaughter ...

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  • 1/ KỂ NHỮNG NGUYÊN NHÂN CỦA NGỘ ĐỘC THỨC ĂN Ngoại độc tố được sản xuất bởi vi sinh vật Các vi sinh vật (vi khuẩn, nấm, virus, ký sinh trùng) (được xếp loại thành xâm nhập (invasive), không xâm nhập (noninvasive) hoặc sinh độc tố (toxin-producing)

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  • NGỘ ĐỘC THỨC ĂN (FOOD POISONING) Phần 2 18/ VI KHUẨN GÂY TIÊU CHẢY NÀO LIÊN KẾT VỚI CO GIẬT DO SỐT Ở TRẺ EM ? Nhiễm trùng Shigella nơi các trẻ nhỏ thường gây nên sốt cao với các cơn co giật do sốt (febrile seizures) trước khi khởi đầu bệnh tiêu chảy. Bệnh có thể rất nặng và tử vong do mất nước (dehydration) có thể xảy ra trong vòng 8 giờ kể từ khi bắt đầu tiêu chảy. Đau bụng và sốt thường đi trước tiêu chảy. Trong trường hợp điển hình, phân có máu và có dạng...

    pdf13p sinhtobo111 13-04-2011 82 8   Download

  • NGỘ ĐỘC THỨC ĂN (FOOD POISONING) Phần 1 1/ KỂ NHỮNG NGUYÊN NHÂN CỦA NGỘ ĐỘC THỨC ĂN - Ngoại độc tố được sản xuất bởi vi sinh vật - Các vi sinh vật ( vi khuẩn, nấm, virus, ký sinh trùng) (được xếp loại thành xâm nhập ( invasive ), không xâm nhập ( noninvasive ) hoặc sinh độc tố ( toxin-producing) - Các chất hiện diện trong thức ăn một cách tự nhiên ( amatoxin (nấm), dinoflagellates hay thallophytes) 2/ MỨC ĐỘ LƯU HÀNH CỦA NGỘ ĐỘC THỨC ĂN? CÓ GÂY CHẾT NGƯỜI KHÔNG? Từ năm 1996 đến 1997, người...

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  • Food Poisoning S. aureus is among the most common causes of food-borne outbreaks of infection in the United States. S. aureus food poisoning results from the inoculation of toxin-producing S. aureus into food by colonized food handlers. Toxin is then elaborated in such growth-promoting food as custards, potato salad, or processed meats. Even if the bacteria are killed by warming, the heat-stable toxin is not destroyed. The onset of illness is rapid, occurring within 1–6 h of ingestion.

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  • Clinical Manifestations Intestinal Disorders Food Poisoning C. perfringens, primarily type A, is the second or third most common cause of food poisoning in the United States (Chap. 122). The responsible toxin is thought to be a cytotoxin produced by 75% of strains isolated from cases of foodborne disease. The cytotoxin binds to a receptor on the small-bowel brush border and induces a calcium ion–dependent alteration in permeability. The associated loss of ions alters intracellular metabolism, resulting in cell death.

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  • B. cereus can produce either a syndrome with a short incubation period— the emetic form, mediated by a staphylococcal type of enterotoxin—or one with a longer incubation period (8–16 h)—the diarrheal form, caused by an enterotoxin resembling E. coli LT, in which diarrhea and abdominal cramps are characteristic but vomiting is uncommon. The emetic form of B. cereus food poisoning is associated with contaminated fried rice; the organism is common in uncooked rice, and its heat-resistant spores survive boiling. If cooked rice is not refrigerated, the spores can germinate and produce toxin.

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  • Loperamide should not be used by patients with fever or dysentery; its use may prolong diarrhea in patients with infection due to Shigella or other invasive organisms. b The recommended antibacterial drugs are as follows: Travel to high-risk country other than Thailand: Adults: (1) A fluoroquinolone such as ciprofloxacin, 750 mg as a single dose or 500 mg bid for 3 days; levofloxacin, 500 mg as a single dose or 500 mg qd for 3 days; or norfloxacin, 800 mg as a single dose or 400 mg bid for 3 days. (2) Azithromycin, 1000 mg as a single dose or 500 mg qd...

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  • Age Most of the morbidity and mortality from enteric pathogens involves children

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  • History The answers to questions with high discriminating value can quickly narrow the range of potential causes of diarrhea and help determine whether treatment is needed. Important elements of the narrative history are detailed in Fig. 122-1. Physical Examination The examination of patients for signs of dehydration provides essential information about the severity of the diarrheal illness and the need for rapid therapy. Mild dehydration is indicated by thirst, dry mouth, decreased axillary sweat, decreased urine output, and slight weight loss.

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  • Host Defenses Given the enormous number of microorganisms ingested with every meal, the normal host must combat a constant influx of potential enteric pathogens. Studies of infections in patients with alterations in defense mechanisms have led to a greater understanding of the variety of ways in which the normal host can protect itself against disease. Normal Flora The large numbers of bacteria that normally inhabit the intestine act as an important host defense by preventing colonization by potential enteric pathogens.

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  • Tham khảo tài liệu 'chapter 122. acute infectious diarrheal diseases and bacterial food poisoning (part 6)', y tế - sức khoẻ, y học thường thức phục vụ nhu cầu học tập, nghiên cứu và làm việc hiệu quả

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  • Pathogenic Mechanisms Enteric pathogens have developed a variety of tactics to overcome host defenses. Understanding the virulence factors employed by these organisms is important in the diagnosis and treatment of clinical disease. Inoculum Size The number of microorganisms that must be ingested to cause disease varies considerably from species to species. For Shigella, enterohemorrhagic Escherichia coli, Giardia lamblia, or Entamoeba, as few as 10–100 bacteria or cysts can produce infection, while 105–108Vibrio cholerae organisms must be ingested orally to cause disease.

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